IFN-γ-Stimulated Transcriptional Activation by IFN-γ-Activated Transcriptional Element-Binding Factor 1 Occurs via an Inducible Interaction with CAAAT/Enhancer-Binding Protein-β

IFN - γ IFN - Metalloproteinase - 9 Gene Expression by Transcriptional Suppression of Matrix

Semi-Quantitative Analysis of HOXA11, Leukemia Inhibitory Factor and Basic Transcriptional Element Binding Protein 1 mRNA Expression in the Mid-Secretory Endometrium of patients with endometriosis

Background: HOXA11 and leukemia inhibitory factor (LIF) and basic transcriptional element binding protein1 (BTEB1) are expressed in endometrium throughout the menstrual cycle and show a dramatic increase during the mid-luteal phase at the time of implantation. In this case-control study, the expression pattern of these mRNA was evaluated in patients with endometriosis at the time of implantatio...

IGF-I regulation by IFN-γ requires STAT-5b TRANSCRIPTIONAL REGULATION OF INSULIN-LIKE GROWTH FACTOR-I (IGF-I) BY INTERFERON-GAMMA (IFN-γ) REQUIRES STAT-5b

IFN - and Posttranscriptional Up - Regulation by γ Offsets Transcriptional Synergism with IFN - Mycobacterium tuberculosis Expression by Posttranscriptional Inhibition of

AMP-activated protein kinase restricts IFN-γ signaling.

Inflammation in the CNS contributes to neurologic disorders. Neuroinflammation involves the release of inflammatory molecules from glial cells, such as astrocytes and microglia, and can lead to neuronal damage if unabated. In multiple sclerosis, peripheral immune cells, including IFN-γ-producing Th1 cells, infiltrate the CNS and are important in shaping the inflammatory microenvironment, in par...